Molecular function of human senataxin protein, mutated in ataxia oculomotor apraxia type 2 (AOA2) and amyotrophic lateral sclerosis type 4 (ALS4), in the pathology of these diseases.

Research Categorization
Researcher and Organization
Principal Investigator
Principal Investigator First Name
Natalia
Principal Investigator Last Name
Gromak
Awardee Organization
Contact PI Country
Project Detail
FY Overall Cost
$11,474
Funding Organization