Project Number 2P50AG005131-26 Agency/Funding Organization NIA Funding Year 2009 View Full Project Details for COGNITIVE PROFILES AND PREDICTORS OF DECLINE IN DLB, PDD, AND AD Research Categorization Primary Disease / Condition Multiple Dementias CADRO Category B. Diagnosis, Assessment, and Disease Monitoring 3. Traditional assessments Category F. Research Resources 1. Alzheimer's Disease Centers Researcher and Organization Principal Investigator SALMON, DAVID P Principal Investigator First Name DAVID P Principal Investigator Last Name SALMON Awardee Organization UNIVERSITY OF CALIFORNIA SAN DIEGO Awardee State California Contact PI Country United States Project Detail Funding Opportunity Announcement RFA-AG-09-001: ALZHEIMER'S DISEASE RESEARCH CENTERS (P50) FY Overall Cost $178,985 Funding Organization Agency/Funding Organization NIA Funding Organization Country United States Program Official PHELPS, CREIGHTON H. Related Resources Repository [AlzPED] CRF receptor-1 antagonism mitigates Aβ pathology and cognitive and synaptic deficits in a mouse model of Alzheimer's disease [AlzPED] Peptides of presenilin-1 bind the amyloid precursor protein ectodomain and offer a novel and specific therapeutic approach to reduce ß-amyloid in Alzheimer's disease [AlzPED] Novel therapeutic strategy for neurodegeneration by blocking Aβ seeding mediated aggregation in models of Alzheimer's disease. [AlzPED] Neural stem cells genetically-modified to express neprilysin reduce pathology in Alzheimer transgenic models. [AlzPED] A Neuroprotective Brain-penetrating Endopeptidase Fusion Protein Ameliorates Alzheimer Disease Pathology and Restores Neurogenesis [AlzPED] Early BDNF treatment ameliorates cell loss in the entorhinal cortex of APP transgenic mice. [AlzPED] Neuroprotective effects of regulators of the glycogen synthase kinase-3beta signaling pathway in a transgenic model of Alzheimer's disease are associated with reduced amyloid precursor protein phosphorylation. [AlzPED] Cerebrolysin decreases amyloid-beta production by regulating amyloid protein precursor maturation in a transgenic model of Alzheimer's disease. [AlzPED] Amelioration of the cerebrovascular amyloidosis in a transgenic model of Alzheimer's disease with the neurotrophic compound cerebrolysin. [AlzPED] The neuroprotective effects of Cerebrolysin in a transgenic model of Alzheimer's disease are associated with improved behavioral performance. Pagination Previous page ‹‹ Page 2
